DIABETES SUGAR

Diabetes Insipidus – exhibiting increased thirst (patients drink from 5 to 25 liters water), polyuria, low relative hydrochloric urine density (1000-1004). Often combined with other endocrine and metabolic disorders and autonomic disorders mi. Forms of diabetes insipidus.                Central diabetes insipidus

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Diabetes Insipidus – exhibiting increased thirst (patients drink from 5 to 25 liters water), polyuria, low relative hydrochloric urine density (1000-1004). Often combined with other endocrine and metabolic disorders and autonomic disorders mi. Forms of diabetes insipidus.                Central diabetes insipidus

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Diffuse toxic goiter (Graves’ disease)

Graves’ disease ( synonyms: Basedow’s disease, Perry, Flajani , Graves ) – an autoimmune disease characterized by diffuse nym thyroid enlargement with considerable tion increase its gormonal Noah activity and leads to intoxication with thyroid hormone – hyperthyroidism. Accurate data on the distribution of diffuse toxic goiter (DTZ) are not available. According to various

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Graves’ disease ( synonyms: Basedow’s disease, Perry, Flajani , Graves ) – an autoimmune disease characterized by diffuse nym thyroid enlargement with considerable tion increase its gormonal Noah activity and leads to intoxication with thyroid hormone – hyperthyroidism. Accurate data on the distribution of diffuse toxic goiter (DTZ) are not available. According to various

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Eye symptoms DIF FUZNOGO toxic goiter

Eye symptoms DIF FUZNOGO toxic goiter result from complex neurohormonal disorders IU mechanism of which to the end ne.raskryt . They usually occur with diffuse toxic goiter. Ocular symptoms diffuse toksichekogo goiter (GSDT3) mainly consist of exophthalmos and disorders associated with eating friendly eye movements GOVERNMENTAL apples (Mobius symptom) and facial muscles due miaste Nia glazodvigatel facial muscles

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Eye symptoms DIF FUZNOGO toxic goiter result from complex neurohormonal disorders IU mechanism of which to the end ne.raskryt . They usually occur with diffuse toxic goiter. Ocular symptoms diffuse toksichekogo goiter (GSDT3) mainly consist of exophthalmos and disorders associated with eating friendly eye movements GOVERNMENTAL apples (Mobius symptom) and facial muscles due miaste Nia glazodvigatel facial muscles

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TREATMENT OF EYE SYMPTOMS OF THYROTOXICOSIS

Treatment of eye symptoms mainly patho genetic. Prevention of exophthalmos consists in timely treatment of hyperthyroidism.                In the presence Prizna Cove exophthalmos is impossible from the start to use large drug doses imidazole, which can lead to TTG hypersecretion, exophthalmic factor.                In what Nation when the euthyroid state must

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Treatment of eye symptoms mainly patho genetic. Prevention of exophthalmos consists in timely treatment of hyperthyroidism.                In the presence Prizna Cove exophthalmos is impossible from the start to use large drug doses imidazole, which can lead to TTG hypersecretion, exophthalmic factor.                In what Nation when the euthyroid state must

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Hypothyroidism (MIXEDEMA)

Hypothyroidism (Myxedema) is a disease caused by the absence or severe insufficiency of thyroid function (Gall 1874, English physician). Hypothyroidism suffer at any age, but most often at the age of 35 : 50 years, women are more likely than

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Hypothyroidism (Myxedema) is a disease caused by the absence or severe insufficiency of thyroid function (Gall 1874, English physician). Hypothyroidism suffer at any age, but most often at the age of 35 : 50 years, women are more likely than

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Hypothyroidism – insufficient formation of thyroid hormones

Hypothyroidism is the most common thyroid dysfunction. In most countries, particularly in Russia, hypothyroidism is due to iodine deficiency. The prevalence of hypothyroidism in the world is 5%. The inability of the thyroid gland to produce enough hormones (T4

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Hypothyroidism is the most common thyroid dysfunction. In most countries, particularly in Russia, hypothyroidism is due to iodine deficiency. The prevalence of hypothyroidism in the world is 5%. The inability of the thyroid gland to produce enough hormones (T4

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CRETINISM

Cretinism – a disease ha characterizes congenital Nedo sufficiency of the thyroid gland , or hypothyroidism athyreosis , retarded growth and mental development, a kind of myxedema swelling. Etiology. The etiology is often caused um brionalnym malformation of the thyroid gland (TG), congenital enzymatic synthesis deficiency of thyroid hormones, congenital goiter on the

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Cretinism – a disease ha characterizes congenital Nedo sufficiency of the thyroid gland , or hypothyroidism athyreosis , retarded growth and mental development, a kind of myxedema swelling. Etiology. The etiology is often caused um brionalnym malformation of the thyroid gland (TG), congenital enzymatic synthesis deficiency of thyroid hormones, congenital goiter on the

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Thyroid adenoma SAME LEZY (Nodular goiter)

ADENOMA THYROID SAME LEZY (nodular goiter) is camping benign tumors of the thyroid. The etiology is not entirely clear. Previous believed that the beginnings of an adenoma of the thyroid gland for imposes in the embryonic zhiz no or early childhood and increase vayutsya under conditions blagopriyatst vuyuschih their development. Factors for

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ADENOMA THYROID SAME LEZY (nodular goiter) is camping benign tumors of the thyroid. The etiology is not entirely clear. Previous believed that the beginnings of an adenoma of the thyroid gland for imposes in the embryonic zhiz no or early childhood and increase vayutsya under conditions blagopriyatst vuyuschih their development. Factors for

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ADDISON’S DISEASE. HYPOCORTICISM

Gipokortitsizm – insufficient accuracy of the adrenal cortex. Would Vaeth primary damage over pochechnikov secondary and from insufficient precision of ACTH secretion by the pituitary gland (adrenocorticotropic hormone). Primary gipokortitsizm may be acute (adrenal apoplexy) and chronic ( Addison bo Leznov). In addition, there may be a latent, erased form of hypocorticism not always recognized due to

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Gipokortitsizm – insufficient accuracy of the adrenal cortex. Would Vaeth primary damage over pochechnikov secondary and from insufficient precision of ACTH secretion by the pituitary gland (adrenocorticotropic hormone). Primary gipokortitsizm may be acute (adrenal apoplexy) and chronic ( Addison bo Leznov). In addition, there may be a latent, erased form of hypocorticism not always recognized due to

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HYPERCORTICISM

Cushing – any cha -particle or mixed increasing function of the adrenal cortex. Hypercorticism can be primary and secondary.                Primary hypercortisolism in the majority of cases, s occurs on the basis corticosteroma and rarely adrenal hyperplasia.                Secondary hypercortisolism arises because of Vyshen hypothalamus AKTG- RF isolation with subsequent higher Niemi ACTH secretion. In

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Cushing – any cha -particle or mixed increasing function of the adrenal cortex. Hypercorticism can be primary and secondary.                Primary hypercortisolism in the majority of cases, s occurs on the basis corticosteroma and rarely adrenal hyperplasia.                Secondary hypercortisolism arises because of Vyshen hypothalamus AKTG- RF isolation with subsequent higher Niemi ACTH secretion. In

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