Category: Thyroid gland diseases

Thyroiditis is based on thyroid inflammation. The following thyroiditis are distinguished: Hashimoto, subacute granulomatous, subacute lymphocytic, Riedel fibrous, purulent. Hashimoto’s thyroiditis Hashimoto’s thyroiditis (autoimmune, lymphomatous struma, lymphadenoid goiter) occurs in most cases in older women with a genetic predisposition due

Riedel’s goiter is a chronic fibrotic process in the thyroid gland. Fibrous thyroiditis can be combined with retroperitoneal and mediastinal fibrosis, primary sclerosing cholangitis, retrobulbar fibrosis, which suggests a common origin. A morphological study reveals proliferation of connective

Hypothyroidism is the most common thyroid dysfunction. In most countries, particularly in Russia, hypothyroidism is due to iodine deficiency. The prevalence of hypothyroidism in the world is 5%. The inability of the thyroid gland to produce enough hormones (T4

It occurs due to the spread of infection from the purulent focus in the middle ear through the veins and sinuses of the temporal bone or as a result of direct contact of pus with

The epiphysis consists of a loose neuroglial stroma that surrounds the nests of epithelial cells containing neurosecretory granules, these cells are called pineocytes. Pineocytes have long thin processes, which are remnants of primitive predecessors of

Difficulties in recognizing hypothyroidism are amplified many times when the disease proceeds in unusual (non-classical) forms. 1.Hypertension in hypothyroidism. Hypertension is detected in 10-30% of patients with hypothyroidism. Its genesis is explained by vessel sensitization to endogenous

The most severe (but fortunately, very rare) complication of hypothyroidism is hypothyroid or myxedematous coma . The basis of its pathogenesis is inhibition of the respiratory center, a progressive decrease in cardiac output, tissue hypoxia and a decrease in

Etiology  Diabetes mellitus develops due to inadequate synthesis and / or secretion of antidiuretic hormone (central form), or the inability of the kidneys to respond adequately to circulating vasopressin (renal form). In endocrinological practice, diabetes

Vascular damage is manifested as a violation of the endothelial relaxing factor and increased leukocyte adhesion to the epithelium surface. Rheological changes associated with increased adhesiveness of platelets are also of great importance in impaired

Isolation genomically second DNA from the venous blood of the patients was conducted meto- house fenolhloroformnoy extraction. Amplification of polymorphic sections was carried out using a polymerase chain reaction on an RNS-2 thermal cycler (manufactured by Techne, United Kingdom). Amplification was